PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 19, entitled, “BMAL1 modulates senescence programming via AP-1.”
Aging (Aging-US) Authors
PRESS RELEASE: A new priority research paper was published on the cover of Aging’s Volume 15, Issue 17, entitled, “Fail-tests of DNA methylation clocks, and development of a noise barometer for measuring epigenetic pressure of aging and disease.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 16, entitled, “Dectin-1 stimulation promotes a distinct inflammatory signature in the setting of HIV-infection and aging.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 15, entitled, “Natural aging and ovariectomy induces parallel phosphoproteomic alterations in skeletal muscle of female mice.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 14, entitled, “Human senescent fibroblasts trigger progressive lung fibrosis in mice.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 10, entitled, “Stress granules sequester Alzheimer’s disease-associated gene transcripts and regulate disease-related neuronal proteostasis.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 9, entitled, “In vitro and in vivo effects of zoledronic acid on senescence and senescence-associated secretory phenotype markers.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 5, entitled, “AAV1.NT-3 gene therapy prevents age-related sarcopenia.”
Capturing the genetic architecture of Alzheimer’s disease (AD) is challenging because of the complex interplay of genetic and non-genetic factors in its etiology. It has been suggested that AD biomarkers may improve the characterization of AD pathology and its genetic architecture…
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 14, Issue 23, entitled, “Single nuclei profiling identifies cell specific markers of skeletal muscle aging, frailty, and senescence.”