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Aging-US.net features press releases on the latest aging research, plus interviews and from the distinguished network of authors who continue to publish their research with Aging-US.

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Aging-US Authors

Figure 7. Autophagy contributes to cells’ survival during chronological aging.
Aging News

Autophagy’s Role in DNA Loss and Survival of Diploid Yeast Cells During Chronological Aging

October 25, 2023October 25, 2023

PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 19, entitled, “Live while the DNA lasts. The role of autophagy in DNA loss and survival of diploid yeast cells during chronological aging.”

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Figure 6. KEGG pathway enrichment analysis of astrocyte differential hub genes suggests a metabolic switch from aerobic glycolysis to oxidative phosphorylation during aging.
Aging News

Integrative Analysis Reveals Metabolic Switch in Aging Astrocytes

October 24, 2023October 24, 2023

PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 19, entitled, “Metabolic switch in the aging astrocyte supported via integrative approach comprising network and transcriptome analyses.”

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Dr. Wenbo Yu, Stanley
Aging News Spotlight

Behind the Study: Poisson Distribution-Based Model of Cancer Rates & Cancer Risk-Dependent Theory of Aging

October 23, 2023

Dr. Wenbo Yu, Stanley from the Centre for Cancer Biology, SA Pathology and University of South Australia details a research perspective he co-authored that was published by Aging (Aging-US) in Volume 15, Issue 17, entitled, “A Poisson distribution-based general model of cancer rates and a cancer risk-dependent theory of aging.”

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Figure 1. Effect of aging on endothelial cell DNA damage.
Aging News

Reducing Double-strand DNA Break Repair Exacerbates Vascular Aging

October 18, 2023

PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 19, entitled, “Reduction of double-strand DNA break repair exacerbates vascular aging.”

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Figure 4. BMAL1 contributes to AP-1 mediated transcriptional control of the senescence program and confers resistance to apoptosis.
Aging News

Senescence Program Modulated by BMAL1 via AP-1

October 16, 2023

PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 19, entitled, “BMAL1 modulates senescence programming via AP-1.”

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Aging Volume 15, Issue 19
Aging Latest Issue

BMAL1 modulates senescence programming via AP-1

October 15, 2023

Cellular senescence and circadian dysregulation are biological hallmarks of aging. Whether they are coordinately regulated has not been thoroughly studied. The researchers hypothesize that BMAL1, a pioneer transcription factor and master regulator of the molecular circadian clock, plays a role in the senescence program…

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Metabolic switch in the aging astrocyte supported via integrative approach comprising network and transcriptome analyses
Aging Latest Issue

Metabolic switch in the aging astrocyte supported via integrative approach comprising network and transcriptome analyses

October 15, 2023
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Reduction of double-strand DNA break repair exacerbates vascular aging
Aging Latest Issue

Reduction of double-strand DNA break repair exacerbates vascular aging

October 15, 2023
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Pathways explaining racial/ethnic and socio-economic disparities in dementia incidence: the UK Biobank study
Aging News

Insights on Disparities in Dementia From the UK Biobank Study

October 11, 2023

PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 18, entitled, “Pathways explaining racial/ethnic and socio-economic disparities in dementia incidence: the UK Biobank study.”

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Figure 1. Experimental workflow and subject demographics. (A) PBMC samples were collected from three young and three older adults (midpoint of the age range shown) before and seven days after vaccination. The samples were negatively enriched for B cells.
Aging News

B Cell Response After Influenza Vaccine in Young and Older Adults

October 10, 2023

PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 18, entitled, “High-throughput single-cell profiling of B cell responses following inactivated influenza vaccination in young and older adults.”

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