Dr. Judith Campisi discusses her priority research paper published in 2021 by Aging, entitled, “Cdkn1a transcript variant 2 is a marker of aging and cellular senescence.”
Search Results for: behind the study
Dr. Michael Lisanti describes his 2020 paper published by Aging, entitled, “COVID-19 and chronological aging: senolytics and other anti-aging drugs for the treatment or prevention of corona virus infection?”
Dr. Gil Atzmon from the Albert Einstein College of Medicine discusses his 2017 study published by Aging, entitled, “The complex genetics of gait speed: genome-wide meta-analysis approach.”
Dr. Marina Antoch from Roswell Park Comprehensive Cancer Center discusses her 2017 study published by Aging, entitled: “Physiological frailty index (PFI): quantitative in-life estimate of individual biological age in mice.”
Dr. Kara Fitzgerald details her publication by Aging, entitled, “Potential reversal of epigenetic age using a diet and lifestyle intervention: a pilot randomized clinical trial”.
Dr. Alex Zhavoronkov discusses his 2020 research paper published by Aging, entitled, “Geroprotective and senoremediative strategies to reduce the comorbidity, infection rates, severity, and lethality in gerophilic and gerolavic infections.”
PRESS RELEASE: A new research paper was published in Aging ‘sVolume 16, Issue 8, entitled, “Characterization of age-associated gene expression changes in mouse sweat glands.”
PRESS RELEASE – A new research paper was published on the cover of Aging (listed by MEDLINE/PubMed as “Aging (Albany NY)” and “Aging-US” by Web of Science), Volume 16, Issue 17 on September 9, 2024, entitled, “The cell rejuvenation atlas: leveraging network biology to identify master regulators of rejuvenation strategies.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 24, entitled, “Mapping of the gene network that regulates glycan clock of ageing.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 23, entitled, “Sirtuin 6 activation rescues the age-related decline in DNA damage repair in primary human chondrocytes.”