It is with great enthusiasm and a deep sense of responsibility that I have assumed the role of Editor-in-Chief of Aging-US, effective January 1, 2025. Throughout my career at the intersection of aging research, cellular senescence, and translational science, I have witnessed the transformative potential that deeper insights into the biology of aging can have on healthspan and overall quality of life. This is why I am honored to guide Aging-US into its next chapter.
Aging Journal
A new research paper was published in Aging (Aging-US) Volume 17, Issue 4, on April 17, 2025, titled “Dietary associations with reduced epigenetic age: a secondary data analysis of the methylation diet and lifestyle study.”
A new research paper was published in Aging (Aging-US) Volume 17, Issue 4, on April 10, 2025, titled “Impact of Factor Xa inhibitors on cardiovascular events in older patients with nonvalvular atrial fibrillation.”
A new research paper was published in Aging (Aging-US) Volume 17, Issue 4, on April 7, 2025, titled “Pharmacological recapitulation of the lean phenotype induced by the lifespan-extending sulfur amino acid-restricted diet.”
A new research paper was published in Aging (Aging-US) Volume 17, Issue 4, on April 4, 2025, titled “Influence of rapamycin on safety and healthspan metrics after one year: PEARL trial results.”
A new research paper was published in Aging (Aging-US) Volume 17, Issue 4, on April 2, 2025, titled “Fisetin ameliorates vascular smooth muscle cell calcification via DUSP1-dependent p38 MAPK inhibition.”
Aging (Aging-US), published by Impact Journals, is pleased to announce its participation at the upcoming Society for Scholarly Publishing (SSP) 47th Annual Meeting. The event will take place from May 28-30, 2025, in Baltimore, Maryland.
Aging (Aging-US) invites submissions for a Special Collection dedicated to the theme of cellular senescence, spanning its basic mechanisms, physiological and pathological functions, and clinical applications.
A new research paper was published in Aging (Aging-US) on April 2, 2025, as the cover of Volume 17, Issue 4, titled “Decreased mitochondrial NAD+ in WRN deficient cells links to dysfunctional proliferation.”
Werner syndrome (WS), caused by mutations in the RecQ helicase WERNER (WRN) gene, is a classical accelerated aging disease with patients suffering from several metabolic dysfunctions without a cure.