PRESS RELEASE: A new research paper was published in Aging’s Volume 16, Issue 13, entitled, “Co-analysis of methylation platforms for signatures of biological aging in the domestic dog reveals previously unexplored confounding factors.”
Aging-US Authors
PRESS RELEASE: A new research paper was published in Aging’s Volume 16, Issue 13, entitled, “Modulating in vitro lung fibroblast activation via senolysis of senescent human alveolar epithelial cells.”
Roles of PEDF in Exercise-induced Suppression of Senescence and its Impact on Lung Pathology in Mice
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 16, Issue 13, entitled, “Roles of pigment epithelium-derived factor in exercise-induced suppression of senescence and its impact on lung pathology in mice.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 16, Issue 12, entitled, “Aging exacerbates oxidative stress and liver fibrosis in an animal model of Down Syndrome.”
PRESS RELEASE: A new editorial paper was published in Aging’s Volume 16, Issue 12, entitled, “Aging retinal pigmented epithelium: omics-based insights into vision decline.”
PRESS RELEASE: A new review was published on the cover of Aging’s Volume 16, Issue 12, entitled, “Senescent cell-derived vaccines: a new concept towards an immune response against cancer and aging?”
PRESS RELEASE: A new editorial paper was published in Aging’s Volume 16, Issue 11, entitled, “Global consensus for sarcopenia.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 16, Issue 11, entitled, “Associations of childhood, adolescence, and midlife cognitive function with DNA methylation age acceleration in midlife.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 16, Issue 11, entitled, “Dietary sucrose determines the regulatory activity of lithium on gene expression and lifespan in Drosophila melanogaster.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 16, Issue 11, entitled, “Mitophagy and cancer: role of BNIP3/BNIP3L as energetic drivers of stemness features, ATP production, proliferation, and cell migration.”