PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 11, entitled, “Short telomeres in alveolar type II cells associate with lung fibrosis in post COVID-19 patients with cancer.”
Aging (Aging-US) Authors
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 10, entitled, “Key elements of cellular senescence involve transcriptional repression of mitotic and DNA repair genes through the p53-p16/RB-E2F-DREAM complex.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 10, entitled, “DNAmFitAge: biological age indicator incorporating physical fitness.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 10, entitled, “Stress granules sequester Alzheimer’s disease-associated gene transcripts and regulate disease-related neuronal proteostasis.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 9, entitled, “Exogenous exposures shape genetic predisposition to lipids, Alzheimer’s, and coronary heart disease in the MLXIPL gene locus.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 9, entitled, “Highly multiplexed immune profiling throughout adulthood reveals kinetics of lymphocyte infiltration in the aging mouse prostate.”
Impact Journals (Aging’s publisher) is proud to participate at the Society for Scholarly Publishing (SSP) 45th Annual Meeting, which convenes May 31–June 2, 2023, in Portland, Oregon.
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 9, entitled, “Increased expression of musashi 1 on breast cancer cells has implication to understand dormancy and survival in bone marrow.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 9, entitled, “In vitro and in vivo effects of zoledronic acid on senescence and senescence-associated secretory phenotype markers.”
PRESS RELEASE: A new editorial paper was published in Aging’s Volume 15, Issue 8, entitled, “The senescence-associated secretory phenotype induces neuroendocrine transdifferentiation.”