PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 23, entitled, “Uncovering a unique pathogenic mechanism of SARS-CoV-2 omicron variant: selective induction of cellular senescence.”
Aging (Aging-US) Authors
SARS-CoV-2 variants are constantly emerging with a variety of changes in the conformation of the spike protein, resulting in alterations of virus entry mechanisms. Solely omicron variants use the endosomal clathrin-mediated entry. Here, researchers investigate the influence of defined altered spike formations to study their impact on premature cellular senescence.
Nectandrin B for Longevity: Nectandrin B (in Nutmeg) Significantly Increases Lifespan of Fruit Flies
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 22, entitled, “Nectandrin B significantly increases the lifespan of Drosophila – Nectandrin B for longevity.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 22, entitled, “Tat-heat shock protein 10 ameliorates age-related phenotypes by facilitating neuronal plasticity and reducing age-related genes in the hippocampus.”
PRESS RELEASE: A new editorial paper was published in Aging’s Volume 15, Issue 22, entitled, “Artificial intelligence for aging research in cancer drug development.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 22, entitled, “Contribution of membrane raft redox signalling to visfatin-induced inflammasome activation and podocyte injury.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 22, entitled, “Chronological aging impacts abundance, function and microRNA content of extracellular vesicles produced by human epidermal keratinocytes.”
The disturbance of intercellular communication is one of the hallmarks of aging. The goal of this study is to clarify the impact of chronological aging on extracellular vesicles (EVs), a key mode of communication in mammalian tissues.