The ovarian microenvironment becomes fibrotic and stiff with age, in part due to increased collagen and decreased hyaluronan. However, the extracellular matrix (ECM) is a complex network of hundreds of proteins, glycoproteins, and glycans which are highly tissue specific and undergo pronounced changes with age.
Aging (Aging-US) Authors
Dr. Dechao Feng from the Department of Urology, Institute of Urology, West China Hospital, Sichuan University, discusses a research paper he co-authored that was published by Aging (Aging-US) in Volume 15, Issue 18, entitled, “Identification of senescence-related lncRNA prognostic index correlating with prognosis and radiosensitivity in prostate cancer patients.”
Dr. Frank Pun, Diana Zagirova, Dr. Anatoly Urban, and Geoffrey Ho Duen Leung from Insilico Medicine Hong Kong Ltd., discuss a research paper they co-authored that was published by Aging (Aging-US) in Volume 15, Issue 18, entitled, “Biomedical generative pre-trained based transformer language model for age-related disease target discovery.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 19, entitled, “Live while the DNA lasts. The role of autophagy in DNA loss and survival of diploid yeast cells during chronological aging.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 19, entitled, “Metabolic switch in the aging astrocyte supported via integrative approach comprising network and transcriptome analyses.”
Dr. Wenbo Yu, Stanley from the Centre for Cancer Biology, SA Pathology and University of South Australia details a research perspective he co-authored that was published by Aging (Aging-US) in Volume 15, Issue 17, entitled, “A Poisson distribution-based general model of cancer rates and a cancer risk-dependent theory of aging.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 19, entitled, “Reduction of double-strand DNA break repair exacerbates vascular aging.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 19, entitled, “BMAL1 modulates senescence programming via AP-1.”