PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 12, entitled, “Age prediction from human blood plasma using proteomic and small RNA data: a comparative analysis.”
Aging (Aging-US) Authors

PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 11, entitled, “Senescence and senotherapies in biliary atresia and biliary cirrhosis.”

PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 11, entitled, “Old-age-induced obesity reversed by a methionine-deficient diet or oral administration of recombinant methioninase-producing Escherichia coli in C57BL/6 mice.”

PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 11, entitled, “Precious1GPT: multimodal transformer-based transfer learning for aging clock development and feature importance analysis for aging and age-related disease target discovery.”

Aging’s publisher, Impact Journals, is sponsoring Team Open Access in the annual cycling event to end cancer: The Ride for Roswell.

PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 11, entitled, “Short telomeres in alveolar type II cells associate with lung fibrosis in post COVID-19 patients with cancer.”

PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 10, entitled, “Key elements of cellular senescence involve transcriptional repression of mitotic and DNA repair genes through the p53-p16/RB-E2F-DREAM complex.”

PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 10, entitled, “DNAmFitAge: biological age indicator incorporating physical fitness.”

PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 10, entitled, “Stress granules sequester Alzheimer’s disease-associated gene transcripts and regulate disease-related neuronal proteostasis.”

PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 9, entitled, “Exogenous exposures shape genetic predisposition to lipids, Alzheimer’s, and coronary heart disease in the MLXIPL gene locus.”