PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 21, entitled, “Longitudinal characterization of behavioral, morphological and transcriptomic changes in a tauopathy mouse model.”
Aging (Aging-US) Authors
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 20, entitled, “Alcohol consumption and epigenetic age acceleration across human adulthood.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 20, entitled, “Tissue immunoexpression of IL-6 and IL-18 in aging men with BPH and MetS and their relationship with lipid parameters and gut microbiota-derived short chain fatty acids.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 20, entitled, “Deciphering reproductive aging in women using a NOD/SCID mouse model for distinct physiological ovarian phenotypes.”
PRESS RELEASE: A new research perspective was published in Aging’s Volume 15, Issue 20, entitled, “Cholinergic centro-cingulate network in Parkinson disease and normal aging.”
PRESS RELEASE: A new priority research paper was published on the cover of Aging’s Volume 15, Issue 20, entitled, “Proteomic quantification of native and ECM-enriched mouse ovaries reveals an age-dependent fibro-inflammatory signature.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 19, entitled, “Live while the DNA lasts. The role of autophagy in DNA loss and survival of diploid yeast cells during chronological aging.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 19, entitled, “Metabolic switch in the aging astrocyte supported via integrative approach comprising network and transcriptome analyses.”
PRESS RELEASE: A new research paper was published in Aging’s Volume 15, Issue 19, entitled, “Reduction of double-strand DNA break repair exacerbates vascular aging.”
PRESS RELEASE: A new research paper was published on the cover of Aging’s Volume 15, Issue 19, entitled, “BMAL1 modulates senescence programming via AP-1.”