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Aging-US.net features press releases on the latest aging research, plus interviews and from the distinguished network of authors who continue to publish their research with Aging-US.

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Aging Journal

Cellular Senescence and Stem Cells Share a More Complex Relationship Than Previously Recognized
Aging News

Cellular Senescence and Stem Cells Share a More Complex Relationship Than Previously Recognized

June 29, 2026June 29, 2026

BUFFALO, NY — June 29, 2026 — A new review was published in Volume 18 of Aging on May 30, 2026, titled “The love and hate relationship between cellular senescence and stemness.”

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Senescent Muscle Cells Send Molecular Messages That May Contribute to Age-Related Muscle Decline
Aging News

Senescent Muscle Cells Send Molecular Messages That May Contribute to Age-Related Muscle Decline

June 3, 2026June 3, 2026

A new research paper was published in Volume 18 of Aging-US on May 15, 2026, titled “Extracellular vesicles released by senescent myoblasts affect recipient cells via miRNA-target interactions.”

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Extending Healthspan Through Public Health and Longevity Medicine
Aging News

Extending Healthspan Through Public Health and Longevity Medicine

May 20, 2026May 20, 2026

A new editorial was published in Volume 18 of Aging-US on May 18, 2026, titled “Public health in the age of longevity interventions: from prevention to system-wide resilience.”

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Precision Anti-Aging Strategies Aim to Target Harmful Senescent Cells While Preserving Beneficial Ones
Aging News

Precision Anti-Aging Strategies Aim to Target Harmful Senescent Cells While Preserving Beneficial Ones

May 15, 2026May 15, 2026

A new review was published in Volume 18 of Aging-US on May 4, 2026, titled “Cellular senescence: from pathogenic mechanisms to precision anti-aging interventions.”

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Stage-Specific Gene Expression Changes Reveal Early Triggers of Cellular Aging
Aging News

Stage-Specific Gene Expression Changes Reveal Early Triggers of Cellular Aging 

April 29, 2026May 4, 2026

A new research paper was published in Volume 18 of Aging-US on April 10, 2026, titled “Stage-dependent transcriptomic changes in human dermal fibroblast senescence model.”

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Polyploidy-Induced Senescence May Drive Aging, Tissue Repair, and Cancer Risk
Aging News

Polyploidy-Induced Senescence May Drive Aging, Tissue Repair, and Cancer Risk

February 18, 2026February 18, 2026

A new editorial was published in Volume 18 of Aging-US on February 8, 2026, titled “Polyploidy-induced senescence: Linking development, differentiation, repair, and (possibly) cancer?”

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How Cell Cycle Status Affects Aging Cell Response to Senolytic Drugs
Aging News

How Cell Cycle Status Affects Aging Cell Response to Senolytic Drugs

September 23, 2025September 23, 2025

A new research paper was published in Volume 17, Issue 8 of Aging-US on August 7, 2025, titled “Senescent cell heterogeneity and responses to senolytic treatment are related to cell cycle status during senescence induction.”

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Aging (Aging-US) Supports Landmark Senescence and Aging Research Events in Rome
Aging News

Aging (Aging-US) Supports Landmark Senescence and Aging Research Events in Rome

August 11, 2025August 11, 2025

Aging (Aging-US) is proud to support a milestone event for the global senescence and aging research community.

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Dr. Judith Campisi
Aging News

Call for Papers: Commemorative Collection Honoring Dr. Judith Campisi

May 1, 2025May 2, 2025

Aging (Aging-US) invites submissions for a Special Collection dedicated to the theme of cellular senescence, spanning its basic mechanisms, physiological and pathological functions, and clinical applications.

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Senolytics Dasatinib and Quercetin for Prevention of Pelvic Organ Prolapse in Mice
Aging News

Senolytics Dasatinib and Quercetin for Prevention of Pelvic Organ Prolapse in Mice

October 22, 2024October 22, 2024

PRESS RELEASE – A new research paper was published in Aging (listed by MEDLINE/PubMed as “Aging (Albany NY)” and “Aging-US” by Web of Science), Volume 16, Issue 19 on September 26, 2024, entitled, “Use of the senolytics dasatinib and quercetin for prevention of pelvic organ prolapse in a mouse animal model.”

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